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Clinical Outcome of Acute Bacterial Prostatitis; A Multicenter Study
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Seong Ju Lee, Jin Mo Koo, Bong Suk Shim, Yong Hyun Cho, Chang Hee Han, Seung Ki Min, Sung Joo Lee, Hwan Cheol Son, Jun Mo Kim, Jong Bo Choi, Tae Hyoung Kim, Sang Kuk Yang, Kil Ho Lee, Yong Kil Na, Sung Ho Lee, Hee Jong Jung, Seung Il Jung, Chul Sung Kim, Jae Min Chung, Young Jin Seo, Won Yeol Cho, Kweon Sik Min, Sang Don Lee
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Korean J Urogenit Tract Infect Inflamm 2011;6(2):165-170. Published online October 31, 2011
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 Abstract
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- "Purpose: Proper guidelines concerning antibiotic administration for acute bacterial prostatitis (ABP) are unclear. We retrospectively analyzed treatment status and clinical outcomes to establish a proper treatment guideline. Materials and Methods: The clinical records of 669 patients from 21 hospitals diagnosed with ABP were reviewed. Prior manipulation, antibiotics administration, mean length of treatment, complication and procedure were analyzed. Results: The mean age of 538 patients (80.4%) without manipulation (group 1) and 131 patients (19.6%) with manipulation (group 2) was 58.3 years (range 19-88 years). Transrectal prostate biopsy was the most common cause of acute bacterial prostatitis (n=66; 50.4%). Of the clinical symptoms in the non-manipulation and manipulation groups, fever was most common (88.2% and 86.3%, respectively). Acute urinary retention (14.3% and 28.1%, respectively) was significantly increased in the manipulation group (p<0.05). Escherichia coli was the most frequently isolated bacterium from urine (72.0% and 66.7% of cases, respectively). Mean length of treatment was 6.5days and 7.9days, respectively; the difference was significant (p<0.05). Combination antibiotic therapy with third generation cephalosporin+aminoglycoside was used in 49.3% and 55.5% of cases, respectively. For single antibiotic therapy, second generation quinolones were used the most (35.5% and 34.3%, respectively). Sequale occurred in 29 group 1 patients (5.4%) and 20 group 2 patients (15.3%); the difference was significant (p<0.05). Conclusions: Prior manipulation was associated with 20% of ABP patients. Regardless of manipulation, clinical outcome was similar after treating with appropriate antibiotics."
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The Effect of Inflammatory Histological Extent of Prostate on the Lower Urinary Tract Symptoms in Benign Prostatic Hyperplasia Patients
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Seong Ju Lee, Dong Hyeon Lee, Young Yo Park, Bongsuk Shim, Woon-Sup Han
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Korean J Urogenit Tract Infect Inflamm 2011;6(1):54-60. Published online April 30, 2011
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Abstract
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- "Purpose: to investigate for clinical correlation between inflammatory histological findings of the prostate and lower urinary tract symptoms in benign prostatic hyperplasia patients. Materials and Methods: A total of 274 patients whose prostate-specific antigen (PSA) was higher than 4.0 ng/dl, had underwent prostate biopsy. International prostate symptom score questionnaire, uroflowmetry, and transrectal ultrasonography were also performed. Patients were divided into 3 groups and granted points according to the extent of lymphocytic infiltration: 0 point for patients with normal findings; 1 point for patients with lower than 50% of lymphocytic infiltration; 2 points for patients with higher than 50% of lymphocytic infiltration or secretor destruction by neutrophil infiltration findings. We quantified the extent of inflammation by using total prostatitis pathology score and classified 0-5 points, 6-10 points, 11-15 points, higher than 16 points into grade 1-4, respectively. Results: Of the 274 patients, 71 who diagnosed with prostate cancer from their biopsy were excluded. Of the remaining 203 patients, 106 (52.21%) were classified grade 1, 57 (28.08%) were grade 2, 31 (15.27%) were grade 3, and 9 (4.43%) were grade 4. There were 142 patients (69.96%) in the group with core 2, which means severe inflammation, and 61 patients (30.04%) in the group without core 2. In addition, prostate volume, storage symptoms score and total scores in IPSS and quality of life were significantly higher in the group with core 2. Conclusions: Lower urinary tract symptoms deteriorated as prostatic inflammation became severe. However, further studies are required to determine correlation more accurately."
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KAUTI
